Sequelae ntawm lub taub hau raug mob

Lawv tuaj yeem sib txawv ntawm ib tus neeg mus rau lwm tus. Nws kwv yees tias 90% ntawm tag nrho cov neeg raug mob taub hau tsis muaj sequelae ntawm lawv cov CD. 5 mus rau 8% tam sim no tseem ceeb sequelae thiab rau 1%, cov sequelae loj heev nrog rau qhov ua tsis taus pa tsis tu ncua.

Ntawm qhov tshwm sim, peb tuaj yeem pom:

  • Mob taub hau
  • Kauj taub hau
  • Tsis meej pem Syndrome
  • A qaug dabpeg, ib txwm ua tau, tsis hais qhov kev siv ntawm lub taub hau raug mob (mob, nruab nrab lossis hnyav). Nws tshwm sim hauv 3% ntawm tag nrho cov neeg mob taub hau.
  • Nyob rau hauv lub sij hawm ntev, muaj kev pheej hmoo ntawm meningitis tshwm sim yog hais tias lub taub hau raug mob nrog ib tug externalized ntws ntawm cerebrospinal kua, tshwj xeeb tshaj yog nyob rau hauv cov pob txha ntawm lub ntsej muag (lub qhov ntswg, pob ntseg, thiab lwm yam).
  • A tuag tes tuag taw, ntau dua los yog tsawg dua, uas nyob ntawm qhov chaw ntawm lub hlwb lesion.
  • cov kev pab cuam rwj cerebral, uas tuaj yeem tshwm sim thaum lub cev txawv teb chaws nkag mus rau hauv lub hlwb, thaum cov pob txha pov tseg los yog yooj yim heev thaum CT nrog rau pob txha taub hau nrog kev nyuaj siab.
  • Ntau yam kev puas tsuaj neuro-sensory (tsis hnov ​​​​lus lossis tsis hnov ​​​​tsw, txo kev ua siab ntev rau qee yam stimuli (suab nrov))
  • Ib qho deterioration ntawm kev txawj ntse thiab lub hlwb ua haujlwm
  • Ib qho poob ntawm kev sib npaug
  • Hais lus nyuaj
  • Ua kom qaug zog
  • Memorization, concentration, comprehension nyuaj…
  • Apathy los yog ntawm qhov tsis sib xws chim siab, impulsiveness, disinhibition, mus ob peb vas ...

Cov sequelae tuaj yeem ua pov thawj rau hauv tsev kho mob hauv lub chaw kho mob rau cov neeg mob hlwb.

Sau ntawv cia Ncua